Where is the lateral ventricles located

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Jones, J. Lateral ventricle. Reference article, Radiopaedia. Central Nervous System. URL of Article. On this page:.

Quiz questions. Stranding S. Gray's anatomy. Churchill Livingstone. Read it at Google Books - Find it at Amazon 2.

Ross LMMP. Atlas of anatomy. George Thieme Verlag. Read it at Google Books - Find it at Amazon 3. Anatomy for Diagnostic Imaging. Related articles: Anatomy: Brain. Promoted articles advertising. Cases and figures. Figure 1: human brain - lateral view Figure 1: human brain - lateral view. Figure 2: Gray's illustration Figure 2: Gray's illustration. The frontal, temporal, and occipital lobes include the anterior, inferior, and posterior horns, respectively.

It connects with the anterior horn at the anterior side at the position of the interventricular foramen. The body eventually approaches the splenium of the corpus callosum. It has a triangular cross-section with a roof, floor, and medial wall, with the top surface layer and bottom surface meeting on the lateral aspects. The septum pellucidum and the body of the fornix, which is shared by two lateral ventricles, create the medial wall. The bottom layer is primarily created medially by the superior layer of the thalamus and on the lateral side by a structure known as the caudate nucleus.

The stria terminalis and thalamostriate veins are located between these two structural components. The tapetum forms the ceiling and lateral walls, whereas the medial wall has two peaks, one inferior and one superior, and is known as the calcar avis.

A feature known as the bulb of the posterior horn is located above those altitudes. The anterior also known as the frontal horn of the lateral ventricle is isolated from the centre half by an imagined vertical path that passes at the position of the interventricular foramen. This addition features a triangle cross-section and a roof, medial wall and a base layer or floor. The far more anterior portion of the corpus callosum trunk forms the roof, whereas the top of the caudate nucleus forms the floor.

The septum pellucidum forms the medial wall. The hippocampus, along with the alveus and fimbriae, composes the majority of the base of the inferior horn. The collateral eminence is uplift in the lateral region of the surface caused by the inner folding of the white mater that lays deep to the collateral sulcus. The lateral section of the roof lateral wall is made up of tapetum fibres, whereas the medial portion is made up of the caudate nucleus tail and the stria terminalis. From the base of the central portion, these components continue through the top layer of the inferior horn.

The caudate nucleus tail and the stria terminalis cease on the anterior side of the amygdaloid complex, which is present in the most anterior portion of the top surface layer of the lateral ventricles.

It goes anteroinferiorly into the temporal lobe from the posterior end of the central area. The inferior horn features a small hollow in cross-section that is bordered on the top and laterally by the top, and beneath it and medially by the base.

This arrangement is the reason why the lateral section of the ceiling is known as the lateral wall, while the medial part of the floor is known as the medial wall. Cerebrospinal fluid is produced by the ventricle covering CSF. The cerebrospinal fluid CSF is then absorbed in the subarachnoid region after passing through the ventricular system.

Cerebrospinal fluid CSF is thought to have several key roles in the brain. It helps to make the brain buoyant, reducing the stress and agony that gravity and movements could otherwise produce. The reality is that if the brain is not maintained in some form of a liquid phase, it will become altered and twisted beneath its bodily mass, and sensitive tissue cells will begin to rip and be harmed.

The barrier of Cerebrospinal fluid CSF that covers the brain also acts as a barrier against potential hazards connected with mechanical stress or any other type of applied force, such as if a person falls. Another example would be, if a person falls and harms their head severely, or if they are beaten and a blunt force is applied to the head. Regardless of pressure fluctuations within the ventricles, the rate of Cerebrospinal fluid CSF production in the ventricles stays constant.

If the flow of Cerebrospinal fluid CSF is impeded at any point or site in the ventricular system, it might be a problem. Cerebrospinal Fluid CSF will continue to be available and produced, but it will be unable to leave the platform. As a result, the stress inside the ventricles would rise, and the increasing pressure may effectively cause the ventricles to enlarge.

The expanding ventricles may then clash with other brain regions, leading to a variety of health complications based on where the blockage occurred and which structures or tissues are most influenced by this expansion. When this occurs in children whose skulls have not fully ossified typically under the age of 2 , the head may enlarge.

When CSF is produced in the lateral ventricle, it fills the cavity before passing through the interventricular foramen of Monro and entering the third ventricle. Cerebrospinal fluid CSF produced in the third ventricle departs the area via the Sylvius cerebral aqueduct and enters the fourth ventricle in addition to CSF produced in the lateral ventricle.

The cerebral ventricular system is made up of these chambers and their interconnected passages. A CT scan can be used to determine the volume of the lateral ventricles and other structures within the brain.

Physicians can use the scan to determine not just the length of the ventricles, but also the density of the cerebrospinal fluid CSF they hold. This data can be utilized to prevent and manage brain disorders such as hydrocephalus, which is an excessive accumulation of fluid in the ventricle.

Ventriculomegaly is a disorder that causes the lateral ventricle to develop improperly. The explanation of ventriculomegaly is uncertain, but it is extremely debated that atrophy of structural components present around the lateral ventricles, as well as a decrease in the amount of the nearby periventricular structures, may be major factors responsible, allowing the ventricles to broaden and fill in the region.

One possibility is a stoppage of venous blood or Cerebrospinal fluid CSF circulation which leads to an increase in the volume of cerebrospinal fluid CSF in the ventricles. A further explanation is that mechanical pressure and shear pressures cause injury and atrophy of surrounding structures that encircle the ventricles.